Mesenchymal actomyosin contractility is required for androgen-driven urethral masculinization in mice
Abstract
The morphogenesis of mammalian embryonic external genitalia (eExG) shows dynamic differences between males and females. In genotypic males, eExG are masculinized in response to androgen signaling. Disruption of this process can give rise to multiple male reproductive organ defects. Currently, mechanisms of androgen-driven sexually dimorphic organogenesis are still unclear. We show here that mesenchymal-derived actomyosin contractility, by MYH10,...
Paper Details
Title
Mesenchymal actomyosin contractility is required for androgen-driven urethral masculinization in mice
Published Date
Mar 8, 2019
Journal
Volume
2
Issue
1
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