Fructose-induced inflammation and increased cortisol: A new mechanism for how sugar induces visceral adiposity
Abstract
Traditionally, the leading hypothesis regarding the development of obesity involves caloric imbalance, whereby the amount of calories consumed exceeds the amount of calories burned which causes obesity. Another hypothesis for why we get fat has surfaced in the last decade which is the idea that the overconsumption of added sugars and refined carbohydrates induce insulin resistance and high insulin levels causing obesity. While insulin is a...
Paper Details
Title
Fructose-induced inflammation and increased cortisol: A new mechanism for how sugar induces visceral adiposity
Published Date
May 1, 2018
Volume
61
Issue
1
Pages
3 - 9
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