Original paper
AT1-receptor-deficiency induced atheroprotection in diabetic mice is partially mediated via PPARγ
Abstract
Peroxisome-proliferator–activated-receptor-γ (PPARγ) acts as a transcriptional regulator of multiple genes involved in glucose and lipid metabolism. In vitro studies showed that activated PPARγ suppresses AT1R-gene expression and vice versa. However, it has not yet been determined in vivo, whether AT1R-PPARγ-interactions play a relevant role in the pathogenesis of diabetic complications and specifically in accelerated atherosclerosis. ApoE−/−...
Paper Details
Title
AT1-receptor-deficiency induced atheroprotection in diabetic mice is partially mediated via PPARγ
Published Date
Jan 1, 2013
Journal
Volume
12
Issue
1
Pages
30 - 30
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Notes
History