The Alternative Oxidase AOX Does Not Rescue the Phenotype oftko25tMutant Flies
Abstract
A point mutation [technical knockout25t (tko25t)] in the Drosophila gene coding for mitoribosomal protein S12 generates a phenotype of developmental delay and bang sensitivity. tko25t has been intensively studied as an animal model for human mitochondrial diseases associated with deficiency of mitochondrial protein synthesis and consequent multiple respiratory chain defects. Transgenic expression in Drosophila of the alternative oxidase (AOX)...
Paper Details
Title
The Alternative Oxidase AOX Does Not Rescue the Phenotype oftko25tMutant Flies
Published Date
Oct 1, 2014
Journal
Volume
4
Issue
10
Pages
2013 - 2021
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