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Journal of Thrombosis and Haemostasis
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A prominent clinical feature of severe Covid-19 infection is respiratory failure associated with pulmonary coagulopathy. Recent reports published in Thrombosis and Haemostasis show that treatment with low-molecular weight heparin (LMWH) decreases mortality in critically ill patients with sepsis-induced hypercoagulation, and thus argue for prophylactic administration of the anticoagulant. In addition, the authors point to non-anticoagulant activities of heparin, in particular anti-inflammatory ef...
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#1Arian Arjomandi Rad (Imperial College London)
#2Robert Vardanyan (Imperial College London)
Last. Natalie R Tas ('KCL': King's College London)
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Recent and developing literature has begun reporting on the incidence of thromboembolic events associated with COVID-19. Klok et al. analysed 184 SARS-CoV2-positive ICU patients in two Dutch University Hospitals, reporting an incidence of thrombotic complications to be 31%, with Pulmonary Embolism (PE) comprising 81% of these complications. [1] Moreover, Cui et al. reports on a population of 81 ICU patients at the Union Hospital, Wuhan, an incidence of 25% in VTE, also possibly related to worse ...
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#1Francisco Sandro Menezes-Rodrigues (UNIFESP: Federal University of São Paulo)H-Index: 4
#2José Gustavo Padrão Tavares (UNIFESP: Federal University of São Paulo)H-Index: 3
Last. Afonso Caricati-Neto (UNIFESP: Federal University of São Paulo)H-Index: 14
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Abstract Most severe manifestations of COVID-19 cases, such as multiple organ failure and death, have been linked to coagulation dysfunction markers, such as platelet reduction and increases in prothrombin time, fibrin degradation products and, mainly, D-dimer [1]. A recent paper by Tang et al. [2] in this journal reported that heparin treatment reduced mortality of COVID-19 patients with elevated D-dimer; similar preliminary results have been reported elsewhere [3]. A mounting body of evidence ...
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Abstract We appreciate the opportunity to respond to the letter from Dr Gris and colleagues. It is true there were several limitations in our study. However, we still believe that D-dimer level at admission could be an effective and easily available predictor in patients with coronavirus disease 2019 (COVID-19).
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#1Pieter M. De Kesel (Ghent University Hospital)
#2Katrien Devreese (UGent: Ghent University)H-Index: 23
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#1Nathan T. Connell (Harvard University)H-Index: 4
#2Elisabeth M. Battinelli (Harvard University)H-Index: 16
Last. Jean-Marie Connors (Harvard University)H-Index: 28
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Abstract Recently in the Journal of Thrombosis and Haemostasis, Harzallah and colleagues report the results of antiphospholipid antibody testing in a series of 56 patients with confirmed or suspected SARS-CoV-2 infection.[1] Twenty-five patients were found to be positive for lupus anticoagulants, while five patients had either anticardiolipin or anti??2-glycoprotein I antibodies. The isotypes for the anticardiolipin and anti??2-glycoprotein I antibodies were reportedly IgG and IgM, although spec...
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#1Ning Tang (HUST: Huazhong University of Science and Technology)H-Index: 1
We appreciate the opportunity to respond to the letter from Dr. Ines Harzallah, et al. Antiphospholipid antibody assays including lupus anticoagulant (LAC), anticardiolipin and anti-beta2-glycoprotein I have also been performed in dozens of our patients, however, very few of them got positive results, we don't think that antiphospholipid antibody exists universally in COVID-19 patients. In addition, two of the three reported cases with antiphospholipid antibodies mentioned in the letter [1] also...
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BACKGROUND: Emicizumab is an alternative non-factor approach for treating patients with hemophilia A. However, there is a potential risk of thrombotic events when emicizumab is concomitantly administered with pro-hemostatic therapies. OBJECTIVES: To assess the hemostatic effect in vitro when a plasma-derived factor VIII concentrate containing von Willebrand factor (pdFVIII/VWF) was added to hemophilia A plasma (HAp) in combination with emicizumab. METHODS: HAp and HAp with FVIII inhibitors (HAp-...
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#1Xiaobo Yang (HUST: Huazhong University of Science and Technology)H-Index: 1
#2You Shang (HUST: Huazhong University of Science and Technology)H-Index: 20
Dr. Jecko Trachil stressed a possibility of platelet consumption to form pulmonary thrombi in patients with DOVID-19(1) , which coincides with the theory, which we are working on right now, that pulmonary thrombi may be responsible for hypoxemia before typical acute respiratory distress syndrome develops(2) , ie silent hypoxemia called by some experts(3) . As for anti-platelet drugs attenuating thrombi formation, the balance would be too delicate to maintain, because they may block the platelet ...
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