1900-P: HNF1A Deficiency Leads to Perturbed Glucagon Secretion in Humans
Abstract
Mutations in HNF1A cause Maturity Onset Diabetes of the Young (HNF1A-MODY). Carriers are normoglycemic in childhood, but with age, they develop hyperglycemia associated with insulin secretory defects and beta cell dysfunction. Although endogenous glucose production has been reported, glucagon in HNF1A-MODY has been less explored. Patients respond well to low dose sulfonylureas such as gliclazide, which increases insulin secretion independent of...
Paper Details
Title
1900-P: HNF1A Deficiency Leads to Perturbed Glucagon Secretion in Humans
Published Date
Jun 1, 2020
Journal
Volume
69
Issue
Supplement_1
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