Original paper
A cell permeant phosphopeptide mimetic of Niban inhibits p38 MAPK and restores endothelial function after injury
Abstract
Vascular injury leads to membrane disruption, ATP release, and endothelial dysfunction. Increases in the phosphorylation of p38 mitogen-activated protein kinase (p38 MAPK) and decreases in the phosphorylation of Niban, a protein implicated in ER stress and apoptosis, are associated with vascular injury. A cell permeant phosphopeptide mimetic of Niban (NiPp) was generated. The effects of NiPp in restoring endothelial function were determined ex...
Paper Details
Title
A cell permeant phosphopeptide mimetic of Niban inhibits p38 MAPK and restores endothelial function after injury
Published Date
May 12, 2020
Journal
Volume
34
Issue
7
Pages
9180 - 9191
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Notes
History