Accelerated osteocyte senescence and skeletal fragility in mice with type 2 diabetes
Volume: 5, Issue: 9
Published: May 7, 2020
Abstract
The worldwide prevalence of type 2 diabetes (T2D) is increasing. Despite normal to higher bone density, patients with T2D paradoxically have elevated fracture risk resulting, in part, from poor bone quality. Advanced glycation endproducts (AGEs) and inflammation as a consequence of enhanced receptor for AGE (RAGE) signaling are hypothesized culprits, although the exact mechanisms underlying skeletal dysfunction in T2D are unclear. Lack of...
Paper Details
Title
Accelerated osteocyte senescence and skeletal fragility in mice with type 2 diabetes
Published Date
May 7, 2020
Volume
5
Issue
9
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