TGF-β3/Smad3 Contributes to Isoflurane Postconditioning Against Cerebral Ischemia–Reperfusion Injury by Upregulating MEF2C

Volume: 40, Issue: 8, Pages: 1353 - 1365
Published: Mar 4, 2020
Abstract
Isoflurane postconditioning alleviates cerebral ischemic–reperfusion injury (CIRI), but the underlying mechanism has not been fully clarified. We previously demonstrated that the transforming growth factor beta-1 (TGF-β1)/Smads signaling pathway is involved in the neuroprotective effect of isoflurane postconditioning. TGF-β3 has a highly homologous sequence relative to that of TGF-β1. In this study, we explored the roles of the TGF-β3/Smad3...
Paper Details
Title
TGF-β3/Smad3 Contributes to Isoflurane Postconditioning Against Cerebral Ischemia–Reperfusion Injury by Upregulating MEF2C
Published Date
Mar 4, 2020
Volume
40
Issue
8
Pages
1353 - 1365
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