Multiple Pathways To Avoid Beta Interferon Sensitivity of HIV-1 by Mutations in Capsid
Abstract
HIV-1 infection causes robust innate immune activation in virus-infected patients. This immune activation is characterized by elevated levels of type I interferons (IFNs), which can block HIV-1 replication. Recent studies suggest that the viral capsid protein (CA) is a determinant for the sensitivity of HIV-1 to IFN-mediated restriction. Specifically, it was reported that the loss of CA interactions with CPSF6 or CypA leads to higher IFN...
Paper Details
Title
Multiple Pathways To Avoid Beta Interferon Sensitivity of HIV-1 by Mutations in Capsid
Published Date
Dec 1, 2019
Journal
Volume
93
Issue
23
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