CRMP2–Neurofibromin Interface Drives NF1-related Pain
Abstract
An understudied symptom of the genetic disorder Neurofibromatosis type 1 (NF1) is chronic idiopathic pain. We used targeted editing of Nf1 in rats to provide direct evidence of a causal relationship between neurofibromin, the protein product of the Nf1 gene, and pain responses. Our study data identified a protein-interaction network with collapsin response meditator protein 2 (CRMP2) as a node and neurofibromin, syntaxin 1A, and the N-type...
Paper Details
Title
CRMP2–Neurofibromin Interface Drives NF1-related Pain
Published Date
Jun 1, 2018
Journal
Volume
381
Pages
79 - 90
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