Persistent kallikrein 5 activation induces atopic dermatitis-like skin architecture independent of PAR2 activity

Volume: 140, Issue: 5, Pages: 1310 - 1322.e5
Published: Nov 1, 2017
Abstract
Upregulation of kallikreins (KLKs) including KLK5 has been reported in atopic dermatitis (AD). KLK5 has biological functions that include degrading desmosomal proteins and inducing proinflammatory cytokine secretion through protease-activated receptor 2 (PAR2). However, due to the complex interactions between various cells in AD inflamed skin, it is difficult to dissect the precise and multiple roles of upregulated KLK5 in AD skin.We...
Paper Details
Title
Persistent kallikrein 5 activation induces atopic dermatitis-like skin architecture independent of PAR2 activity
Published Date
Nov 1, 2017
Volume
140
Issue
5
Pages
1310 - 1322.e5
Citation AnalysisPro
  • Scinapse’s Top 10 Citation Journals & Affiliations graph reveals the quality and authenticity of citations received by a paper.
  • Discover whether citations have been inflated due to self-citations, or if citations include institutional bias.