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Aging and cancer in transgenic and mutant mice.

Published on Jan 1, 2003in Frontiers in Bioscience2.21
· DOI :10.2741/1122
Vladimir N. Anisimov2
Estimated H-index: 2
Abstract
1. Abstract 2. Introduction 3. Genetically modified mouse models of accelerated aging 3.1. Growth hormone (GH) transgene mice 3.2. Senescence accelerated mice (SAM) 3.3. Mutation in mouse klotho gene 3.4. DNA repair gene transgenic and knockout models 3.5. Overexpression of Cu, Zn-SOD or catalase 3.6. Mutant and transgenic models of the immunosenescence 3.7. Transgenic and knockout models of age-related neurodegenerative diseases 3.8. Knockout p53 mice 3.9. Regulation of cell-to-cell communication and knockout mouse models 3.10. Telomerase transfected and knockout mice 3.11. HER-2/neu transgenic mice 3.12. Circadian clock gene models 4. Genetically modified mouse models of postponed aging 4.1. Ames dwarf mice 4.2. Growth hormone receptor knockout mice 4.3. Insulin/IGF-1 signaling pathway modifications and longevity in mice 4.4. Knockout p66 gene mice 4.5. O-methylguanine-DNA methyltransferase (MGMT) transgenic mice 4.6. Thioredoxin transgenic mice 4.7. Urokinase plasminogen activator (alpha-MUPA) transgenic mice 5. Conclusion 6. Acknowledgment 7. References
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