Exogenous NAD + supplementation protects H 9c2 cardiac myoblasts against hypoxia/reoxygenation injury via S irt1‐p53 pathway
Abstract
Nicotinamide adenine dinucleotide (NAD(+) ) not only transfers electrons in mitochondrial respiration, but also acts as an indispensable cosubstrate for Sirt1, the class III histone/nonhistone deacetylase. However, NAD(+) is depleted in myocardial ischemia/reperfusion (IR) injury. The objective of this study was to investigate the role of exogenous NAD(+) supplementation in hypoxia/reoxygenation (HR)-stressed H9c2 cardiac myoblasts. Firstly, the...
Paper Details
Title
Exogenous NAD + supplementation protects H 9c2 cardiac myoblasts against hypoxia/reoxygenation injury via S irt1‐p53 pathway
Published Date
Feb 6, 2013
Volume
28
Issue
2
Pages
180 - 189
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