Inefficient degradation of cyclin B1 re-activates the spindle checkpoint right after sister chromatid disjunction
Abstract
Sister chromatid separation creates a sudden loss of tension on kinetochores, which could, in principle, re-activate the spindle checkpoint in anaphase. This so-called “anaphase problem” is probably avoided by timely inactivation of cyclin B1-Cdk1, which may prevent the spindle tension sensing Aurora B kinase from destabilizing kinetochore–microtubule interactions as they lose tension in anaphase. However, exactly how spindle checkpoint...
Paper Details
Title
Inefficient degradation of cyclin B1 re-activates the spindle checkpoint right after sister chromatid disjunction
Published Date
Jun 17, 2014
Journal
Volume
13
Issue
15
Pages
2370 - 2378
Citation AnalysisPro
You’ll need to upgrade your plan to Pro
Looking to understand the true influence of a researcher’s work across journals & affiliations?
- Scinapse’s Top 10 Citation Journals & Affiliations graph reveals the quality and authenticity of citations received by a paper.
- Discover whether citations have been inflated due to self-citations, or if citations include institutional bias.
Notes
History