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2012 Revised International Chapel Hill Consensus Conference Nomenclature of Vasculitides

Published on Jan 1, 2013in Arthritis & Rheumatism9.002
· DOI :10.1002/art.37715
J. C. Jennette3
Estimated H-index: 3
(UNC: University of North Carolina at Chapel Hill),
Ronald J. Falk73
Estimated H-index: 73
(UNC: University of North Carolina at Chapel Hill)
+ 25 AuthorsRichard A. Watts50
Estimated H-index: 50
(UEA: University of East Anglia)
Sources
Abstract
2012 Revised International Chapel Hill Consensus Conference Nomenclature of Vasculitides J. Jennette;R. Falk;P. Bacon;N. Basu;M. Cid;F. Ferrario;L. Flores-Suarez;W. Gross;L. Guillevin;E. Hagen;G. Hoffman;D. Jayne;C. Kallenberg;P. Lamprecht;C. Langford;R. Luqmani;A. Mahr;E. Matteson;P. Merkel;S. Ozen;C. Pusey;N. Rasmussen;A. Rees;D. Scott;U. Specks;J. Stone;K. Takahashi;R. Watts; Arthritis & Rheumatism
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Here we discuss recent advances in understanding the biochemical, immunologic, and genetic pathogenesis of IgA nephropathy, the most common primary glomerulonephritis. Current data indicate that at least four processes contribute to development of IgA nephropathy. Patients with IgA nephropathy often have a genetically determined increase in circulating levels of IgA1 with galactose-deficient O-glycans in the hinge-region (Hit 1). This glycosylation aberrancy is, however, not sufficient to induce...
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