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#1Carol Dobson-Stone (UNSW: University of New South Wales)H-Index: 34
#2Marianne Hallupp (NeuRA: Neuroscience Research Australia)H-Index: 16
Last. Thomas Fath (Macquarie University)H-Index: 19
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Frontotemporal dementia and amyotrophic lateral sclerosis are clinically and pathologically overlapping disorders with shared genetic causes. We previously identified a disease locus on chromosome 16p12.1-q12.2 with genome-wide significant linkage in a large European Australian family with autosomal dominant inheritance of frontotemporal dementia and amyotrophic lateral sclerosis and no mutation in known amyotrophic lateral sclerosis or dementia genes. Here we demonstrate the segregation of a no...
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#1Rowena Kong (UBC: University of British Columbia)
The functional roles of ventromedial prefrontal cortex and amygdala in affecting emotional processing in decision-making have been raised in support of the somatic marker hypothesis. However, latter studies demonstrated challenges to such support based on preserved cognition in the form of reversal learning in ventromedial prefrontal cortex damaged patients tested with a shuffled variant of Iowa Gambling Task. This finding provides implications for cognitive neglect in somatic marker hypothesis ...
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#1P. H. R. SilvaH-Index: 2
Last. Renata F. Leoni (USP: University of São Paulo)H-Index: 8
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Human cognition and behavior emerge from neuronal interactions on a brain structural architecture. The convergence (or divergence) between functional dynamics and structural connectivity and their relationship with cognition is still a pivotal question about the brain. We focused on the information processing speed (IPS), assessed by the Symbol Digit Modalities Test (SDMT), once delayed IPS underlies attention deficits in various clinical conditions. We hypothesize that the structural connectivi...
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#1W Richard Bevan-Jones (University of Cambridge)H-Index: 4
#2Thomas E. Cope (Cognition and Brain Sciences Unit)H-Index: 1
Last. Robert Arnold (University of Cambridge)H-Index: 16
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The work was supported by National Institute for Health Research Cambridge Biomedical Research Centre including the Cambridge Brain Bank; the Wellcome Trust (JBR, 103838); the Cambridge Centre for Parkinson-plus; the Medical Research Council (MR/P01271X/1); the National Institute for Health Research, Association of British Neurologists and the Patrick Berthoud Charitable Trust (TEC); and the Lundbeck Foundation (SSK).
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