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Ibrahim M. Sayed
University of California, San Diego
4Publications
1H-index
2Citations
Publications 4
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#1Ibrahim M. Sayed (UCSD: University of California, San Diego)H-Index: 1
#2Amal A. Elkhawaga (Assiut University)
Last.Mohamed A. El-Mokhtar (Assiut University)H-Index: 4
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Abstract Hepatitis E virus (HEV) is the most common cause of acute viral hepatitis globally. HEV belongs to the Hepeviridae family and at least five genotypes (gt) infect humans. Several animal species are reservoirs for different HEV strains, and they are the source of infection for humans. Some HEV strains are species specific, but other strains could cross species and infect many hosts. The study of HEV infection and pathogenesis was hampered due to the lack of an in vitro and in vivo robust ...
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#1Ibrahim M. Sayed (UCSD: University of California, San Diego)H-Index: 1
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#1Ibrahim M. Sayed (UCSD: University of California, San Diego)H-Index: 1
2 CitationsSource
#1Ayse Z Sahan (UCSD: University of California, San Diego)H-Index: 1
#2Tatiana Venkova (UTMB: University of Texas Medical Branch)H-Index: 1
Last.Soumita Das (UCSD: University of California, San Diego)H-Index: 20
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The accumulation of Helicobacter pylori infection-induced oxidative DNA damage in gastric epithelial cells is a risk factor for developing gastric cancer (GC); however, the underlying mechanisms remain poorly understood. Here we report that the suppression of NEIL2, an oxidized base-specific mammalian DNA glycosylase, is one such mechanism via which H. pylori infection may fuel the accumulation of DNA damage during the initiation and progression of GC. Using a combination of cultured cell lines ...
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