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Laura E.R. Blok
Radboud University Nijmegen
ASDAutismAutism spectrum disorderHabituationIntellectual disability
4Publications
1H-index
5Citations
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Publications 4
Newest
#1Michaela Fenckova (Radboud University Nijmegen)H-Index: 10
#2Laura E.R. Blok (Radboud University Nijmegen)H-Index: 1
Last. Annette Schenck (Radboud University Nijmegen)H-Index: 31
view all 14 authors...
Abstract Background Although habituation is one of the most ancient and fundamental forms of learning, its regulators and its relevance for human disease are poorly understood. Methods We manipulated the orthologs of 286 genes implicated in intellectual disability (ID) with or without comorbid autism spectrum disorder (ASD) specifically in Drosophila neurons, and we tested these models in light-off jump habituation. We dissected neuronal substrates underlying the identified habituation deficits ...
1 CitationsSource
#1Michaela Fenckova (Radboud University Nijmegen)H-Index: 10
#2Laura E.R. Blok (Radboud University Nijmegen)H-Index: 1
Last. Annette Schenck (Radboud University Nijmegen)H-Index: 31
view all 14 authors...
Abstract Background Although habituation is one of the most ancient and fundamental forms of learning, its regulators and relevance for human disease are poorly understood. Methods We manipulated the orthologs of 286 genes implicated in intellectual disability (ID) with or without comorbid autism spectrum disorder (ASD) specifically in Drosophila neurons, and tested these models in light-off jump habituation. We dissected neuronal substrates underlying the identified habituation deficits and int...
1 CitationsSource
#1Michaela Fenckova (Radboud University Nijmegen)H-Index: 10
#2Lenke AsztalosH-Index: 2
Last. Annette Schenck (Radboud University Nijmegen)H-Index: 31
view all 12 authors...
Background: Although habituation is one of the most ancient and fundamental forms of learning, its regulators and relevance for human disease are poorly understood. Methods: We manipulated the orthologs of 286 genes implicated in intellectual disability (ID) with or without comorbid autism spectrum disorder (ASD) specifically in Drosophila neurons, and tested these models in light-off jump habituation. We dissected neuronal substrates underlying the identified habituation deficits and integrated...
2 CitationsSource
#1Michaela Fenckova (Radboud University Nijmegen)H-Index: 10
#2Lenke AsztalosH-Index: 2
Last. Annette Schenck (Radboud University Nijmegen)H-Index: 31
view all 12 authors...
Background: Although habituation is one of the most ancient and fundamental forms of learning, its regulators and relevance for human disease are poorly understood. Methods: We manipulated the orthologs of 286 genes implicated in intellectual disability (ID) with or without comorbid autism spectrum disorder (ASD) specifically in Drosophila neurons, and tested these models in light-off jump habituation. We dissected neuronal substrates underlying the identified habituation deficits and integrated...
1 CitationsSource
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