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A. Björklund
Karolinska Institutet
4Publications
3H-index
45Citations
Publications 4
Newest
#1M Veitenhansl (LMU: Ludwig Maximilian University of Munich)H-Index: 1
#2K Stegner (LMU: Ludwig Maximilian University of Munich)H-Index: 1
Last.H Sekihara (LMU: Ludwig Maximilian University of Munich)H-Index: 1
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13 CitationsSource
#1A. Björklund (KI: Karolinska Institutet)H-Index: 3
#2J. Bondo Hansen (Novo Nordisk)H-Index: 11
Last.Valdemar Grill (KI: Karolinska Institutet)H-Index: 45
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Aims/hypothesis We tested whether chronic overstimulation by levels of hyperglycaemia commonly found in Type 2 diabetes can irreversibly desensitise beta cells and, if so, whether desensitisation relates to the reduction of insulin content and/or the number of beta cells.
14 CitationsSource
#1Arne MelanderH-Index: 51
#2Julie OlssonH-Index: 6
Last.M. CummingsH-Index: 2
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2 CitationsSource
#1A. Björklund (KI: Karolinska Institutet)H-Index: 3
#2G. Yaney (BMC: Boston Medical Center)H-Index: 1
Last.Gordon C. Weir (Joslin Diabetes Center)H-Index: 85
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at the Third JDF World Conference on Diabetes Research held in Monaco in 1991 there was little, if any, mention of the role of fatty acids in pancreatic betacell function. However, this situation has now changed dramatically on the heels of two recent and simultaneousdevelopments. The first has to do with the notion that there is at work within the beta cell, just as in other tissues, an important element of glucose-fatty acid “cross-talk” which appears to be essential for the normal regulation ...
16 CitationsSource
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