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A. Björklund
Karolinska Institutet
4Publications
3H-index
37Citations
Publications 4
Newest
Published on Aug 1, 2004in Diabetologia 7.11
M Veitenhansl1
Estimated H-index: 1
(LMU: Ludwig Maximilian University of Munich),
K Stegner1
Estimated H-index: 1
(LMU: Ludwig Maximilian University of Munich)
+ 5361 AuthorsA Findlow1
Estimated H-index: 1
(University of Salford)
Published on May 1, 2004in Diabetologia 7.11
A. Björklund3
Estimated H-index: 3
(KI: Karolinska Institutet),
J. Bondo Hansen11
Estimated H-index: 11
(Novo Nordisk)
+ 1 AuthorsValdemar Grill46
Estimated H-index: 46
(KI: Karolinska Institutet)
Aims/hypothesis We tested whether chronic overstimulation by levels of hyperglycaemia commonly found in Type 2 diabetes can irreversibly desensitise beta cells and, if so, whether desensitisation relates to the reduction of insulin content and/or the number of beta cells.
Published on Aug 1, 1999in Diabetologia 7.11
Arne Melander50
Estimated H-index: 50
,
Jane E. Olsson5
Estimated H-index: 5
+ 4508 AuthorsEvans Jmm.1
Estimated H-index: 1
Published on Mar 1, 1997in Diabetologia 7.11
A. Björklund3
Estimated H-index: 3
(KI: Karolinska Institutet),
G. Yaney1
Estimated H-index: 1
(BMC: Boston Medical Center)
+ 1 AuthorsGordon C. Weir83
Estimated H-index: 83
(Joslin Diabetes Center)
at the Third JDF World Conference on Diabetes Research held in Monaco in 1991 there was little, if any, mention of the role of fatty acids in pancreatic betacell function. However, this situation has now changed dramatically on the heels of two recent and simultaneousdevelopments. The first has to do with the notion that there is at work within the beta cell, just as in other tissues, an important element of glucose-fatty acid “cross-talk” which appears to be essential for the normal regulation ...
1