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Joshua A. Boyce
Harvard University
214Publications
48H-index
7,692Citations
Publications 214
Newest
Published on 2019in Allergy6.77
Joshua A. Boyce48
Estimated H-index: 48
(Brigham and Women's Hospital)
Published on Apr 12, 2019in American Journal of Respiratory and Critical Care Medicine16.49
Katherine N. Cahill1
Estimated H-index: 1
(Vandy: Vanderbilt University),
Katherine N. Cahill1
Estimated H-index: 1
(Vandy: Vanderbilt University)
+ 6 AuthorsTanya M. Laidlaw19
Estimated H-index: 19
(Harvard University)
Rationale: Daily high-dose aspirin therapy benefits many patients with aspirin-exacerbated respiratory disease but provides no benefit for aspirin-tolerant asthmatics. Type 2 inflammation character...
Published on May 1, 2019
D. Foer (Brigham and Women's Hospital), S.H. Baek (Brigham and Women's Hospital)+ 5 AuthorsJoshua A. Boyce48
Estimated H-index: 48
(Brigham and Women's Hospital)
Published on May 1, 2019in Mucosal Immunology7.35
Tao Liu9
Estimated H-index: 9
(Brigham and Women's Hospital),
Nora A. Barrett14
Estimated H-index: 14
(Harvard University)
+ 7 AuthorsJoshua A. Boyce48
Estimated H-index: 48
(Harvard University)
Cysteinyl leukotrienes (cysLTs) facilitate eosinophilic mucosal type 2 immunopathology, especially in aspirin-exacerbated respiratory disease (AERD), by incompletely understood mechanisms. We now demonstrate that platelets, activated through the type 2 cysLT receptor (CysLT2R), cause IL-33-dependent immunopathology through a rapidly inducible mechanism requiring the actions of high mobility box 1 (HMGB1) and the receptor for advanced glycation end products (RAGE). Leukotriene C4 (LTC4) induces s...
Published on Mar 1, 2019in The Journal of Allergy and Clinical Immunology
Dingxin Pan1
Estimated H-index: 1
(Harvard University),
Kathleen M. Buchheit6
Estimated H-index: 6
(Harvard University)
+ 4 AuthorsJoshua A. Boyce48
Estimated H-index: 48
(Harvard University)
Background Classical FceRI-induced mast cell (MC) activation causes synthesis of arachidonic acid (AA)–derived eicosanoids (leukotriene [LT] C 4 , prostaglandin [PG] D 2 , and thromboxane A 2 ), which mediate vascular leak, bronchoconstriction, and effector cell chemotaxis. Little is known about the significance and regulation of eicosanoid generation in response to nonclassical MC activation mechanisms. Objectives We sought to determine the regulation and significance of MC-derived eicosanoids ...
Joshua A. Boyce48
Estimated H-index: 48
(Brigham and Women's Hospital)
Published on Feb 1, 2019in The Journal of Allergy and Clinical Immunology
Katherine L. Tuttle2
Estimated H-index: 2
,
Alex R. Mola1
Estimated H-index: 1
+ 4 AuthorsPatrick J. Brennan85
Estimated H-index: 85
Published on Feb 1, 2019in The Journal of Allergy and Clinical Immunology
Katherine N. Cahill9
Estimated H-index: 9
(Harvard University),
Katherine Murphy3
Estimated H-index: 3
(Brigham and Women's Hospital)
+ 3 AuthorsTanya M. Laidlaw19
Estimated H-index: 19
(Harvard University)
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