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Haesook T. Kim
Harvard University
ImmunologyHematopoietic stem cell transplantationGraft-versus-host diseaseTransplantationMedicine
345Publications
57H-index
10.6kCitations
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Publications 342
Newest
#1Eduardo Espada (Harvard University)H-Index: 1
#2Matthew P. Cheng (Harvard University)H-Index: 10
Last. Mahasweta Gooptu (Harvard University)H-Index: 4
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Clinical disease caused by BK virus reactivation is a frequent complication of allogeneic hematopoietic cell transplantation (HCT). Because of the lack of effective antiviral agents, BK virus-specific T cells are emerging as a potential therapy for BK virus disease, but the immune response to BK virus after allogeneic HCT has not been well characterized. Our study describes reconstitution of BK virus-specific T-cell immunity in 77 adult patients after HCT. All patients had urinary symptoms, and ...
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#1Benedetta Rambaldi (UNIPV: University of Pavia)
#1Benedetta RambaldiH-Index: 5
Last. Edwin P. AlyeaH-Index: 72
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Haploidentical hematopoietic cell transplantation (haplo-HCT) with post-transplant cyclophosphamide (PTCy) as graft versus host disease (GVHD) prophylaxis is frequently used for patients who do not have HLA-identical donors. However, the effect of PTCy on immune reconstitution (IR) has not been studied extensively in large prospective cohorts, despite a high incidence of early viral infections. We quantified IR in 60 patients after haplo-HCT with PTCy, mycophenolate mofetil and tacrolimus (TAC) ...
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#1Clinton W. Lewis (Auckland City Hospital)
#2Haesook T. Kim (Harvard University)H-Index: 57
Last. Vincent T. Ho (Harvard University)H-Index: 71
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ABSTRACT fludarabine with intravenous busulfan at doses of 3.2 mg/kg (Flu/Bu1) or 6.4mg/kg (Flu/Bu2) Hepatic veno-occlusive disease/sinusoidal obstruction syndrome (VOD/SOS) is a serious complication of hematopoietic stem cell transplantation (HCT) that is felt to be triggered, at least in part, by damage to the liver sinusoidal endothelium from cytotoxic conditioning regimens. Accordingly, the incidence of VOD/SOS after reduced-intensity conditioning (RIC) HCT is low compared with myeloablative...
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#1H. Moses Murdock (Harvard University)
#2Haesook T. Kim (Harvard University)H-Index: 57
Last. R. Coleman Lindsley (Harvard University)H-Index: 22
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#1Cindy Lynn Hickey (Harvard University)
#2Haesook T. Kim (Harvard University)H-Index: 57
Last. Corey Cutler (Harvard University)H-Index: 73
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#1Lindsey E. Roeker (MSK: Memorial Sloan Kettering Cancer Center)H-Index: 7
#2Jennifer R. Brown (Harvard University)H-Index: 54
Last. Anthony R. Mato (MSK: Memorial Sloan Kettering Cancer Center)H-Index: 25
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#1Cindy Lynn Hickey (Harvard University)
#2Haesook T. Kim (Harvard University)H-Index: 57
Last. Rizwan Romee (Harvard University)H-Index: 18
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#1Jacqueline S. Garcia (Harvard University)H-Index: 8
#2Haesook T. Kim (Harvard University)H-Index: 57
Last. Joseph H. Antin (Harvard University)H-Index: 101
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1 CitationsSource
#1Theodoros Karantanos (Johns Hopkins University)H-Index: 17
#2Haesook T. Kim (Harvard University)H-Index: 57
Last. Vassiliki A. Boussiotis (BIDMC: Beth Israel Deaconess Medical Center)H-Index: 56
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#1Theodoros Karantanos (Johns Hopkins University)H-Index: 17
#2Haesook T. Kim (Harvard University)H-Index: 57
Last. Vassiliki A. Boussiotis (BIDMC: Beth Israel Deaconess Medical Center)H-Index: 56
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Abstract BK virus (BKV), a human polyomavirus that remains latent in renal epithelial cells, can be reactivated after hematopoietic stem cell transplantation (HSCT) leading to hemorrhagic cystitis. The incidence of BK viremia is higher after Umbilical cord blood transplantation (UCBT) than HSCT from adult donors. Data regarding the role of immune recovery after UCBT in BKV reactivation is lacking. We examined the correlation between the development of BK viremia and immune reconstitution in 27 a...
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